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exposed to elevated blood pressure. The authors note that this is the first study to find a relationship between sleep and coronary artery calcium deposits. Other studies have looked at sleep apnea -- periods during sleep when an individual stops breathing.Sleep apnea is associated
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Second, inflammation elsewhere in the body increases the risk of calcification, and this effect can be influenced by the body's natural anti-inflammatory hormone, cortisol. This hormone is released in sync with a person's sleep patterns. Increased time asleep might increase the release of cortisol -- which, in turn, might reduce inflammation and, therefore, calcium deposits.
Third, blood pressure declines during sleep. Thus, it might be the case that the longer people sleep, the less their heart is with high blood pressure and heart attacks, but the authors did not measure this sleep phenomenon.
The authors conclude that "this study demonstrates that objectively measured sleep is inversely associated with coronary artery calcification. This study further demonstrates the utility of a simple objective measure of sleep that can be used at Home. Future studies will be needed for crucial extensions to these results